Mohs Rickettsia (r.moseri) is endemic typhus pathogen.
(A) the biological characteristics
Mohs Rickettsia Rickettsia response than Platts in shape, dyeing, resistance and ease of cells susceptible animals and Platts Rickettsia similar, but Mohs Rickettsia-induced guinea pig scrotum body caused stronger.
(B) the pathogenicity and immunity
Clinical features are also endemic typhus epidemic typhus with similar, but milder, shorter duration.
Mohs dissemination Platts Rickettsia Rickettsia different. Mohs Rickettsia long-term latent infection in mice parasitic body, after the rat rat fleas suck blood rickettsial into its digestive tract and multiply in the intestinal epithelial cells. After the release of cell rupture Rickettsia, mixed with flea feces, spread among the group of rats and Mus musculus. Only left mouse turned rat flea bite suck human blood in rats after death, and people infected. So when someone human parasitic lice can be spread by people in the crowd lice secondary ground. In addition, dry flea feces with Rickettsia can be oral, nasal and conjunctival enter the body and cause disease.
Mohs Rickettsia infection with Rickettsia Platts cross-immunity.
(C) Diagnosis and Prevention
Microbiological examination principles and Platts Rickettsia basically the same. After inoculation of guinea pigs from 5 to 6:00, can be observed in guinea pigs, fever, scrotal swelling, testicular smears can be found in the cytoplasm of Rickettsia. Serologic tests have outside Fiji's reaction and complement fixation test.
Prevention is mainly pay attention to hygiene, lice, fleas, rodent. Vaccination can improve the body's immunity.
Chloramphenicol, tetracycline therapy. |
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