Comprehensive transient amnesia (tga), refers to a sudden short-term patients can not accept new information, while retaining the long-term memory, often within 24 hours of relief of a syndrome.
This disease usually without aura symptoms, usually sudden on_set_, mainly memory loss, can not be short of new knowledge, there are varying degrees of retrograde amnesia. Much memory and immediate memory and good, insight and personality, with no advanced cortical dysfunction, the patient verbal exchanges is good, correctly calculated. However, there may be time disorientation, and sometimes go out and coincided with the on_set_ of the patient, often because they can not remember the family address the "missing" for several hours. As the patient developed transient dysfunction of new information, then repeat the question so often due to patients, family members or friends were found. A clear sense of when the on_set_, complex daily activities, such as food, clothes, phone calls, driving, playing musical instruments can be completed. Some patients may also experience headaches, dizziness, tinnitus, feeling abnormal vertebral body basilar artery insufficiency symptoms. Symptoms generally last for several hours, the patient fully recovered after the on_set_ of memory, only the experience of attack can not think of, to form a "memory hole." This is a forgotten episode left the patient the only symptoms, but also the main basis for clinical diagnosis. Physical examination, patients often have no signs of nervous system orientation.
tga true etiology and pathogenesis is unknown, the current majority view, not the epilepsy-like performance, but due to vascular mechanisms. Choroidal artery or the former may be related to cerebral artery transient ischemic cause the central parts of the temporal lobe and the limbic system on hypothalamic vascular ischemia. However, the literature a lot of controversy.
tga benign course, generally no special treatment. There are risk factors for cerebrovascular disease who choose anti-platelet agents, EEG abnormalities were added with anti-epileptic drugs. With cerebral vasospasm were given antispasmodic agents, such as calcium antagonists and so on. Patients within 1 month of on_set_ is best not to engage in complex work. |
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